Akash Mitra

Tagline:CSIR-Senior Research Fellow working in skeletal muscle biology at Yenepoya Research Centre

Mangalore, Karnataka, India

personal photo of Akash Mitra

About Me

I am a PhD student 👨🏻‍🎓 delving into the fascinating world of skeletal muscle 💪🏻 biology and bioactive lipids. Beyond the lab, I immerse myself in the pages 📖 of fantasy novels and psycho thrillers, always eager for an adventure or a twisty plot. I enjoy honing my skills in R programming 🧑🏻‍💻, navigating data with an intermediate flair. As a tech enthusiast, I find joy in the seamless innovation of Apple products. My journey is one of curiosity, exploration, and a love for learning!
I love coffee as well! ☕️

Education

  • Ph.D

    from: 2022, until: present

    Field of study:Skeletal muscle biologySchool:Stem Cells and Regenerative Medicine Centre, Yenepoya Research Centre, Yenepoya UniversityLocation:Mangalore, Karnataka, India

  • M.Sc

    from: 2018, until: 2020

    Field of study:MicrobiologySchool:Adamas UniversityLocation:Kolkata, West Bengal, India

  • B.Sc

    from: 2015, until: 2018

    Field of study:MicrobiologySchool:Adamas UniversityLocation:Kolkata, West Bengal, India

Research Interests

  • Skeletal muscle
  • Muscle atrophy
  • Muscle Stem cells
  • Cell Signalling
  • Inflammation

Publications

  • Quercetin and Kaempferol Mitigate Endotoxin-Induced Skeletal Muscle Wasting by Inhibiting KLF15 Expression and Restoring the Antioxidant System.

    Journal ArticlePublisher:FASEB journal : official publication of the Federation of American Societies for Experimental BiologyDate:2025
    Authors:
    Samanwita MandalAkash MitraBipasha BoseSudheer Shenoy P
    Description:
    The antioxidant properties of quercetin and kaempferol, individually and in combination, were evidenced to attenuate LPS-induced cellular oxidative stress and promote myogenesis by downregulating inflammatory regulators and atrophy-related genes triggered by LPS.
    DOI

  • Unlocking the Potential of Obestatin: A Novel Peptide Intervention for Skeletal Muscle Regeneration and Prevention of Atrophy.

    ReviewPublisher:Molecular biotechnologyDate:2024
    Authors:
    Akash MitraSamanwita MandalBipasha BoseSudheer Shenoy P
    Description:
    There are quite some evidences which points to the fact that obestatin can be purposed as a peptide intervention to prevent skeletal muscle wasting and induce myogenesis to prevent skeletal muscle atrophy and sarcopenia.
    DOI

  • Mitigation of chronic glucotoxicity-mediated skeletal muscle atrophy by arachidonic acid.

    Journal ArticlePublisher:Life sciencesDate:2023
    Authors:
    Akash MitraShanooja ShanavasDebajit ChaudhuryBipasha BoseUndurti N. DasSudheer Shenoy P
    Description:
    A substantial increase in the pro-inflammatory cytokines and ROS production in hyperglycemic conditions, mitigated by AA supplementation and the results imply that AA has cytoprotective actions against hyperglycemia-induced cytotoxicity.
    DOI

  • The elusive role of myostatin signaling for muscle regeneration and maintenance of muscle and bone homeostasis.

    ReviewPublisher:Osteoporosis and sarcopeniaDate:2023
    Authors:
    Akash MitraR. QaisarBipasha BoseShenoy P Sudheer
    Description:
    In this review, the role and regulation of myostatin as a function of muscle regeneration extrapolated to multiple aspects of its molecular functions is put together.
    DOI

  • Sodium fluoride induces skeletal muscle atrophy via changes in mitochondrial and sarcomeric proteomes

    Journal ArticlePublisher:PLOS ONEDate:2022
    Authors:
    Apoorva H NagendraAnimikh RayDebajit ChaudhuryAkash MitraA. RanadeBipasha BoseSudheer Shenoy P
    Description:
    Differential expression of (sarcomeric) proteins, downregulation of 26S proteasome subunits, and differential expression in proteins related to the ubiquitin-proteasome pathway lead to muscle atrophy.
    DOI